NUR 5203 Module 2 Respiratory Case Analysis Example

Reviewed by Junia Fairbank, MSN, RN · American College of Education · Updated

Our NUR 5203 Module 2 example is a respiratory case analysis of a delayed pneumothorax after subclavian central line placement, tracing each assessment finding to the pleural physics that produced it. It answers the second module of American College of Education NUR 5203 (NUR5203, Advanced Pathophysiology and Health Assessment) in ACE's MSN Nurse Educator track. The paper tells the composite case with times, including a clear first chest film and the student's response, and sets the procedure's known risk from McGee and Gould. It explains how negative pleural pressure holds the lung open, traces the needle puncture to air entry, collapse and shunt, explains why oxygen helps little, and describes tension physiology, using Leigh-Smith and Harris to correct a common teaching error. Each finding is paired with its cause, the BTS guideline from Roberts and colleagues frames management, and teaching points close it. Sections usually assign the case.

CourseNUR 5203 Advanced Pathophysiology and Health Assessment
ModuleModule 2
Paper typeRespiratory case analysis
Length1,150 words, about 4 pages plus title and reference pages
FormatAPA 7 student paper
SchoolAmerican College of Education
ProgramMSN Nurse Educator
UpdatedOctober 2026

Free sample paper for NUR 5203 Module 2

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Air Where the Lung Should Be: A Delayed Pneumothorax After Subclavian Line Placement, With Each Assessment Finding Traced to the Pleural Physics That Produced It

Student Name

American College of Education

NUR5203: Advanced Pathophysiology and Health Assessment

Module 2 Assignment

Instructor Name

January 18, 2028

What this page is doingThe title names the complication, the procedure that caused it and the approach of the analysis, finding by finding. The APA 7 title page carries the course line and the module assignment as listed.
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The Case

A 68-year-old composite patient on a medical unit needed a central venous catheter for parenteral nutrition after a complicated bowel resection. A subclavian catheter was placed at the bedside at 1015 after two attempts, and a chest radiograph at 1100 was read as showing correct tip position and no pneumothorax. At 1430 she told the nursing student caring for her that she felt a little short of breath. Her oxygen saturation had fallen from 96% to 91% on room air and her respiratory rate had risen from 16 to 24. The student raised the head of the bed, applied oxygen by nasal cannula and documented anxiety. At 1545 her saturation was 88% on oxygen, her heart rate 118 and her breath sounds were quieter on the right. A repeat radiograph showed a large right pneumothorax. A chest tube was placed, her lung re-expanded, and she recovered.

Central venous catheterization carries a known risk of pneumothorax. McGee and Gould (2003), reviewing the complications of the procedure, reported pneumothorax rates of 1.5% to 3.1% with the subclavian approach, compared with 0.1% to 0.2% with the internal jugular approach. A pneumothorax may not be visible on the first film if air leaks slowly through a small puncture. Film technique matters too. On an upright film, free air rises to the apex and outlines the edge of the lung, but a portable film taken with the patient lying flat or partly reclined lets air collect in front of the lung, where it is much harder to see. A clear early film therefore answers a narrower question than it seems to: it shows that no large pneumothorax was present at that moment, in that position, not that the lung is safe for the rest of the day. Nurses caring for a patient after the procedure are the ones who will see a slow leak declare itself, usually through the patient's breathing long before a second film is ordered.

What this page is doingThe case is told with times and findings, including the student's response, and the procedure's known risk is established from a source. The puzzle for the analysis is a clear first film followed by deterioration.
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How the Pleural Space Holds the Lung Open

The lungs are elastic and, left to themselves, would recoil inward and collapse. The chest wall tends to spring outward. The two are held together by the pleural space, a thin film of fluid between the visceral pleura covering the lung and the parietal pleura lining the chest wall. Because the lung pulls inward and the chest wall pulls outward, the pressure in this space is slightly below atmospheric pressure, typically a few centimeters of water below zero at rest, and it becomes more negative during inspiration as the diaphragm descends. That negative pressure is what keeps the lung expanded against its own recoil. It is also what makes the pleural space dangerous to breach: any opening to the outside or into the airways lets air flow in, because air moves from higher to lower pressure.

What this page is doingNormal pleural mechanics are explained first, because the whole mechanism depends on the negative pleural pressure.
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From a Needle Puncture to a Collapsed Lung

During the second attempt at line placement, the needle almost certainly punctured the apex of the right lung, where the subclavian vein lies close to the pleura. Each breath afterward drew a small amount of air from the lung into the pleural space. As air accumulated, pleural pressure rose toward atmospheric, and the lung, no longer held open, recoiled inward. Because the leak was small and slow, the first radiograph taken 45 minutes after the procedure showed too little air to see. By the afternoon, enough had collected to collapse much of the right lung.

A collapsed lung still receives blood flow from the pulmonary arteries, but the collapsed alveoli receive no air. Blood flowing past those airless alveoli stays deoxygenated, a form of shunt, and this blood mixes with oxygenated blood from the other lung, lowering arterial oxygen levels. Hypoxic vasoconstriction in the collapsed lung partly limits the shunt, which is why saturation often falls gradually rather than all at once. The brain senses falling oxygen and rising effort, and breathing becomes faster. The patient's first complaint, a little short of breath, was the earliest outward sign of a lung that had already begun to collapse.

What this page is doingThe mechanism moves from the puncture to air entry, loss of negative pressure, collapse and shunt, and explains why the first film was clear and why oxygen saturation fell gradually.
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When a Pneumothorax Becomes Tension

In some cases the tear in the lung acts as a one-way valve: air enters the pleural space during inspiration but cannot leave during expiration. Pressure in the affected side then rises above atmospheric, compressing the lung completely and pushing the mediastinum toward the other side. The rising pressure also compresses the great veins and reduces blood returning to the heart, so that cardiac output falls. This is a tension pneumothorax, and it is immediately life threatening.

Nurses are often taught to look for tracheal deviation and hypotension, but these are late findings. Leigh-Smith and Harris (2005), reviewing the evidence, concluded that in patients who are breathing on their own, respiratory distress, tachycardia and falling oxygen saturation are the consistent early features, while hypotension and tracheal deviation tend to appear late, if at all. The case patient's rising heart rate at 1545, together with falling saturation on oxygen, meant that her pneumothorax was large and may have been beginning to affect her circulation.

What this page is doingTension physiology is explained from the one-way valve mechanism, and a review is used to correct a common teaching error about which findings come first.
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Each Finding and Its Cause

Diminished breath sounds on the right: sound from the airways is poorly transmitted through air in the pleural space, and the collapsed lung moves little air. Hyperresonance to percussion on the right: air in the pleural space resonates more than lung tissue. Reduced chest expansion on the right: the collapsed lung no longer inflates, so that side of the chest moves less. Tachypnea and falling saturation: shunt through the collapsed lung and the drive to breathe faster. Tachycardia: compensation for hypoxemia and, as pressure rises, for falling venous return. Anxiety: a real symptom of hypoxemia and breathlessness, not an alternative explanation for them. Current guidance on pleural disease recommends that management be guided by symptoms and physiological effect rather than by size on the radiograph alone (Roberts et al., 2023).

What this page is doingEach finding is paired with its physiological cause in a compact, reusable format that a student could learn from. The final sentence links assessment to current management guidance.
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What Students Need to Be Taught

Three teaching points follow. First, a procedure's known complications define what to watch for afterward. Students should be taught that after a subclavian line, new breathlessness is pneumothorax until proven otherwise, and that a clear first film does not rule out a slow leak. Second, anxiety is a finding to be explained, not an explanation. The student documented anxiety and missed the chance to compare breath sounds side to side. In the author's clinical group, students now practice a two-minute focused respiratory assessment that always compares both sides for sounds, percussion and expansion. Third, the classic signs of tension are late. Students should escalate on rising respiratory rate, falling saturation and rising heart rate, without waiting for hypotension or a shifted trachea. The next module turns from a single condition to two conditions that present alike and must be separated by their mechanisms.

What this page is doingTeaching points come directly from what happened in the case, including a specific skill students will practice, which keeps the educator's perspective the course requires.
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References

Leigh-Smith, S., & Harris, T. (2005). Tension pneumothorax: Time for a re-think? Emergency Medicine Journal, 22(1), 8-16. https://doi.org/10.1136/emj.2003.010421

McGee, D. C., & Gould, M. K. (2003). Preventing complications of central venous catheterization. New England Journal of Medicine, 348(12), 1123-1133. https://doi.org/10.1056/NEJMra011883

Roberts, M. E., Rahman, N. M., Maskell, N. A., Bibby, A. C., Blyth, K. G., Corcoran, J. P., Edey, A., Evison, M., de Fonseka, D., Hallifax, R., Harden, S., Lawrie, I., Lim, E., McCracken, D. J., Mercer, R., Mishra, E. K., Nicholson, A. G., Noorzad, F., Opstad, K., ... Sivasothy, P. (2023). British Thoracic Society guideline for pleural disease. Thorax, 78(Suppl. 3), s1-s42. https://doi.org/10.1136/thorax-2022-219784

Reading the NUR 5203 Module 2 instructions

Module 2 of NUR 5203 often asks you to analyze a respiratory case and link each assessment finding to its physiological cause. Most prompts expect a case summary, normal respiratory mechanics, the mechanism of the disorder, an explanation of each finding and current evidence on recognition or management. Many sections ask what the case teaches students, since the course prepares educators. A paper of about five pages is typical. Your Canvas page names the cases or conditions allowed, whether a table of findings and causes is expected, and whether the case may come from your own practice with details changed.

How this NUR 5203 Module 2 example is built

The analysis begins with the case: a central line placed, a first film read as clear, and hours later rising breathlessness and falling oxygen saturation, with the student's response described factually. The normal mechanics section explains how negative pleural pressure keeps the lung expanded. The mechanism section moves from the needle puncture to slow air entry, loss of negative pressure, partial collapse and shunt, and explains why the first film was clear and why extra oxygen helps little. The tension section explains the one-way valve and uses a review to show that low oxygen and fast breathing appear before tracheal deviation. A compact findings table pairs each sign with its cause, and the last section draws teaching points and a skill to practice.

Where the points sit in the NUR 5203 Module 2 rubric

Respiratory case papers are scored on mechanism, findings and teaching value. Normal mechanics earn credit when explained first, since the disorder makes no sense without them. The mechanism row rewards a complete chain from cause to collapse, including why early imaging can miss it. The tension section earns graduate credit when it corrects a common error with evidence rather than repeating textbook order. Findings marks require each sign paired with its cause, and a compact table helps. Teaching points drawn from what happened in the case, described without blaming anyone, earn the final substantive credit. Current guidelines and APA 7 complete the grade. A findings table that pairs each sign with its mechanism is easy for a grader to check and for students to study.

NUR 5203 Module 2 help: mistakes that cost points

Respiratory papers most often list signs of pneumothorax and stop there. Another frequent weakness is repeating the old teaching that tracheal deviation is an early sign of tension. Students also blame the nurse in the case instead of drawing teaching points. Start with pleural pressure. Explain why the first film can be clear. Pair every finding with its mechanism. Use current guidelines for management statements. Keep the tone factual. If your case is pulmonary embolism, ARDS or an asthma exacerbation, the desk can write a respiratory case analysis on it. Draw the pressure changes as a simple diagram if your section allows figures.

Write yours, or have the desk draft it

This paper is an original model document written by our desk, not a submitted student paper and not an official American College of Education document. Read it for the moves, then write your own to the instructions in your classroom. If you want one built to your exact prompt and rubric, the first custom sample is free and arrives in 24 to 48 hours.

More NUR 5203 and MSN Nurse Educator sample papers

NUR 5203 Module 2 questions, answered

How do I explain respiratory findings by mechanism?

Start with normal ventilation and the pressures involved, then explain what changes in the condition, and link each finding, such as breath sounds, percussion note or oxygen saturation, to the specific change that produces it.

Should I include what the nurse did wrong in the case?

Describe what happened factually and use it to identify teaching points, without blaming an individual. The course's educator focus makes these missed cues valuable learning material.

Where can I find a free NUR 5203 Module 2 sample paper?

The pneumothorax paper is posted whole above at no cost: case, pleural pressure, collapse, tension, a findings table and teaching points.

Why can a chest film be clear right after a line placement?

A small air leak can take hours to collect enough air to show on a film. That is why reassessment after the procedure, not only the first image, matters for a delayed pneumothorax.

What is the earliest sign of tension pneumothorax?

In awake patients, worsening shortness of breath, fast breathing and falling oxygen saturation usually come first. Tracheal deviation and low blood pressure tend to be late signs.